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Despite these many known details, much of the risk for schwannomatosis remained unexplained going into the existing study. Several research organizations had proposed that other schwannomatosis-predisposing genes existed, but nobody had found any. Specializing in genetic research for all forms of the neurofibromatoses, the UAB Medical Genomics Laboratory thought we would focus its analysis on a subset of schwannomatosis samples that didn’t harbor SMARCB1 mutations, which framed their experiments in a way that the part of LZTR1 was revealed. We’ve been working urgently to identify the genetic mechanisms behind these diseases because doing so is central to initiatives to comprehend schwannoma tumor development as well as to identify new prescription drugs, said Ludwine Messiaen, Ph.D., director of the Medical Genomics Laboratory, professor in the Division of Clinical Genetics in the Division of Genetics within the UAB College of Medicine and corresponding study writer.